MOLD Toxicity CAUSING Depression Anxiety Insomnia ADDICTION
At Sponaugle Wellness, Dr Rick Sponaugle is proving that many patients develop alcoholism, OxyContin addiction or Xanax addiction while attempting to self-medicate the following disorders caused by mold toxicity.
Dr. Sponaugle was recently informed by the mold toxicity lab in Dallas that he is the top mold toxicity doctor in North America.
The lab director, Dr. Hooper, was amazed that Dr. Sponaugle could diagnose mold toxicity with a 98 percent accuracy when other physicians using their testing average only 10 percent accuracy.
Dr. Sponaugle explained that he has correlated the brain scans of mold toxic patients with their brain chemistry patterns and mold toxin levels.
Similar patterns occur in patients suffering Benzene toxicity from the Gulf Oil Spill which is less common than mold toxicity.
Dr. Sponaugle also explained that he had personally suffered mold toxicity, Trichothecene toxicity, which has enhanced his ability to recognize the mold toxicity patient.
Dr. Sponaugle further explained that he has been correlatelating brain neurotransmitter profiles with other indirect biomarkers of mold toxicity for years.
Among these biomarkers – elevated immune markers [C3 A and C4 A ] and severe central hormonal suppression [pituitary hormones] including the following; Growth Hormone, MSH, ADH, TSH, ACTH, FSH and LH.
Dr. Sponaugle says that many patients suffering opiate addiction and Xanax addiction use drugs like Oxycontin and Xanax to “calm their anxious brains.”
These patients normally test positive for HLA-DRBQ genetics, genetics that disallow effeicient removal of mold toxins from their body and brain.
Supposedly only 24 percent of Americans have HLA-DRBQ genetics, however, Dr. Sponaugle diagnoses this genetic disorder in 80 percent of his patients.
This genetic abberation is found more commonly in patients of Irish, English, Scandinavian, German essentially northern european descent.
The immune system of patients with HLA-DRBQ genetics does not “tag” fatty toxins as abnormal or “foreign” to the body, hence, these patients do not efficiently remove mold toxins from their brain and body.
In fact, patients with HLA- DRBQ genetics remove mold toxins and other fatty toxins, 468 percent less efficiently, than other patients.
While the symptoms of mold toxicity are many, Dr. Sponaugle says specific symptoms are the driving force as causation of drug addiction and or alcoholism. Initially, patients notice insomnia, then they develop anxiety as well as insomnia. As the brain accumulates higher levels of the mold toxins, Glutamate and PEA, two very strong electrifying brain chemicals, become elevated producing excessive electrical activity in the brain, worst case scenarios develop bipolar symptoms.
Patients self-medicate their brain’s upregulated “electrical voltage” with calming drugs like alcohol, Oxycontin like medication and benzodiazepines such as Xanax and Klonopin.
Dr. Sponaugle has treated many mold toxic patients whose glutamate levels were so high that they exibited bipoolar symptoms and in fact, were sadly diagnosed bioplar by unknowing psychiatrists before coming to Dr. Sponaugle’s clinic. Two young male patients were, in fact, misdiagnosed by University of South Florida psychiatrists as schizophrenic, the university psychiatrists were, of course, naive regarding mold toxicity.
Fortunately, Dr. Steven Stahl of Scripts University in San Diego, recently wrote in his new book on Bipolar and Schizophrenia about the possibility of excessive glutamate, not just excessive dopamine, being the cause of these disorders. While Dr. Stahl did not make the connection with excessive accumulation of mold toxins as the causation of excessive glutamate production, it is a step in the right direction.
Dr. Sponaugle has found excessive glutamate levels in 9 of 10 patients previously diagnosed bipolar, a condition felt by most to be derived from excessive dopamine production, hence, the majority of the medications for Bipolar are dopamine blockers. Dr. Sponaugle recently treated a paranoid 22 year old male who had been misdiagnosed by University of Miami Psychiatrists as bipolar and schizophrenic. When Michael failed to get better after six psychiatric admissions to the University of Miami Psych ward, his parents brought him to Florida Detox for Dr. Sponaugle’s evaluation.
Michael’s history consisted of living in a mold infested apartment on Miami Beach for just six months before he began to develop paranoia. He tested positive for high levels of the Trichothecene Black Mold toxin and Ochratoxin which is produced by Aspergillous Mold. Michael quickly responded to Dr. Sponaugle’s intravenous toxin removal program, his paranoia abated after just one week and after three weeks of treatment his depression and anxiety were gone.*
Because mold toxins shut down hormonal production in the brain’s pituitary gland, patients suffer severe depression and severe chronic fatigue. Pure adrenaline [epinephrine] can not activate it’s receptors in the brain and body without cortisol, cortisol deficiency is common in mold toxic patients. Dopamine can not activate the D2 dopamine receptor [happy receptor] in the brain’s pleasure center with0ut adequate testosterone and thyroid hormone, both of these hormones are deficient in mold toxic patients.
Patients who suffer mold toxicity utilize opiate pain pills for the opiate induced dopamine hit in their dopamine deprived pleasure center, this temporarily treats their depression. They also benefit from the relaxing “calcium channel blockade” effect of opiate pain pills which brings quiescence to their “over electrified” brain.
While mold toxic patients experience severe depression and fatigue, they also feel like their brain is “hot wired” or plugged into an electrical outlet. Over time, as the mold toxicity worsens, patients begin to feel, from a physical perspective, as though they have a 25 pound cement block attached to each leg. Their body feels weak, lethargic and in slow motion, yet, their brain is overelectrified, anxious, irritable and often they exibit rage.